ID | 116306 |
タイトル別表記 | Neurovascular Compression in Atypical Odontalgia Patients
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著者 |
Kawasaki, Kaoru
Tokyo Medical and Dental University|Ohu University
Sugawara, Shiori
Tokyo Medical and Dental University
Watanabe, Kazuya
Tokyo Medical and Dental University
Hong, Chaoli
Tokyo Medical and Dental University
Tu, Trang Thi Huyen
Tokyo Medical and Dental University
Sakamoto, Junichiro
Tokyo Medical and Dental University
Yoshino, Norio
Tokyo Medical and Dental University
Suga, Takayuki
Tokyo Medical and Dental University
Mikuzuki, Lou
Tokyo Medical and Dental University
Takenoshita, Miho
Tokyo Medical and Dental University
Takada, Satoshi
Ohu University
Kurabayashi, Tohru
Tokyo Medical and Dental University
Toyofuku, Akira
Tokyo Medical and Dental University
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キーワード | Persistent Idiopathic Facial Pain (PIFP)
Atypical Odontalgia (AO)
Neurovascular Compression (NVC)
Trigeminal Nerve
Neuropathic Pain
Functional Somatic Symptom
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資料タイプ |
学術雑誌論文
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抄録 | Background. Persistent idiopathic facial pain (PIFP) is the unexplained pain along the territory of the trigeminal nerve, including nonorganic tooth pain called atypical odontalgia (AO). Though PIFP is debilitating to patients’ livelihood and well-being, its pathophysiology remains poorly understood. Although neurovascular compression (NVC) of the trigeminal nerve is known to be associated with trigeminal neuralgia (TN), the relationship between NVC and other orofacial pains has not been fully elucidated. Methods. In this study, we investigated the differences in the characteristics of PIFP (primarily AO) patients in the presence or absence of NVC. A retrospective analysis was performed on data from 121 consecutive patients who had been diagnosed with unilateral PIFP according to the criteria of the International Classification of Headache Disorders (ICHD)–3 and underwent magnetic resonance imaging scans of the head. Results. In the group without NVC, characteristic findings were significant for psychiatric morbidity, somatization, and pain disability, when compared with the group with NVC. Furthermore, the group without NVC exhibited significant headache, noncardiac chest pain, shortness of breath, and pain catastrophizing. Conclusions. These results suggest that PIFP patients can be divided into two groups: one consistent with a neuropathic pain phenotype when NVC is present and a functional somatic symptom phenotype when presenting without NVC. Our findings may enable a more precise understanding of pathophysiology of PIFP and lead to better treatment strategies.
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掲載誌名 |
Pain Medicine
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ISSN | 15264637
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cat書誌ID | AA11706356
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出版者 | American Academy of Pain Medicine|Faculty of Pain Medicine ANZCA|Oxford University Press
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巻 | 21
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号 | 4
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開始ページ | 814
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終了ページ | 821
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発行日 | 2020-02-10
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権利情報 | This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contactjournals.permissions@oup.com
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言語 |
eng
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著者版フラグ |
出版社版
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部局 |
医学系
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